by Gertrud U. Rey It is well known that stress and exposure to UV radiation can reactivate replication of latent herpes simplex virus type 1 and/or type 2 (HSV-1 and/or HSV-2), and the painful lesions associated with these infections. But why is that? During a primary infection, HSV-1 and HSV-2 replicate within the epithelial cells …
The TWiVumvirate discuss the giant Tupanvirus, with the longest tail in the known virosphere, and dampened STING dependent interferon activation in bats. <span data-mce-type=”bookmark” style=”display: inline-block; width: 0px; overflow: hidden; line-height: 0;” class=”mce_SELRES_start”></span><span data-mce-type=”bookmark” style=”display: inline-block; width: 0px; overflow: hidden; line-height: 0;” class=”mce_SELRES_start”></span> Click arrow to play Download TWiV 484 (53 MB .mp3, 87 min) Subscribe (free): iTunes, RSS, email …
TWiV 484: Float like a mimivirus STING like a bat Read More »
On the latest episode of the science show This Week in Virology, a swarm of virologists discusses testing of a MERS coronavirus vaccine for camels, and how a neuronal stress pathway reactivates herpes simplex virus. You can find TWiV #369 at www.microbe.tv/twiv.
The Liberian man who was diagnosed with Ebola virus infection after traveling to Dallas, Texas, was treated with an antiviral drug called brincidofovir. This drug had originally been developed to treat infections with DNA-containing viruses. Why was it used to treat an Ebola virus infection? Brincidofovir (illustrated) is a modified version of an antiviral drug …
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